Lilit, reaguj!

[džin tonik]

austrian superheroes, distribucija - morava.

morava - ash

[lilit]

well, we all know that vienna rules, a pošto sam dobre volje, evo ti i muzička podloga

http://www.youtube.com/watch?v=WZQaEv4bgp4

[Meho Krljic]

Bar smo ovo rešili:

New Test Spots Human Form of Mad Cow Disease with 100 Percent Accuracy

[lilit]

stvarno odlična vest posebno što su nam prioni su i dalje fascinantntan oblik postojanja. niđe života a uzrokuje infekciju.

on a lighter note, upravo se upoznajem  sa alexom!
"alexa, schalte das licht aus, bitte."

[Meho Krljic]

    HPV ima u šteku trikove za koje mi, obični ljudi nismo bili ni svesni da postoje.
  A rare disease gave him tree branch-like hands. After 16 surgeries, he can finally see his fingers.

When the warts first appeared in his teens, speckling his hands and feet, Abul Bajandar wasn't concerned.

Cutting away the unsightly growths himself was too painful, he told AFP, so Bajandar sought medication from a homeopath and herbal specialist in his poor, coastal Bangladeshi village. That just made them worse.

But it wasn't until his early 20s, after he'd worked for years as a rickshaw driver and married his wife, that the warts took on a bark-like appearance and began to rapidly multiply.

By age 25, his hands looked like an overgrown bramble.

People began calling him "Tree Man."

He could not work, or feed himself, or brush his own teeth. Bajandar was in pain. His family, which had consulted doctors in India but couldn't afford treatment, was desperate for answers.
 
So when a television station was in their village covering municipal elections, Bajandar approached them for help, reported the Daily Star.

"At first I thought he was asking for money," Sunil Das, SATV bureau chief, told the Daily Star. "But when he showed me his hands I realized I had never seen anything like that before."

Soon, the man's story became an international sensation, inspiring monetary donations from around the globe and a team of doctors and plastic surgeons at Dhaka Medical College Hospital to treat him free of charge.

"I want to live like a normal person," Bajandar told CNN last February. "I just want to be able to hold my daughter properly and hug her."

Doctors diagnosed the man with epidermodysplasia verruciformis, a rare genetic disease that makes people especially susceptible to human papillomavirus and developing skin tumors. According to AFP, Bajandar was only one of four people in the world to be diagnosed with the illness, which subjects its victims to a lifetime of growths.
  In this report from March 2016, Abul Bajandar begins his round of surgeries that will eventually give him back his hands. The rare disease covered his hands with tree-bark like growths. (Reuters)
In this report from March 2016, Abul Bajandar begins his round of surgeries that will eventually give him back his hands. The rare disease covered his hands with tree-bark like growths. Bangladeshi 'tree man' gets surgery (Reuters) 
In January 2016, surgeons at the hospital performed tests on Bajandar's warts to ensure they could be removed without damaging his major nerves or causing other health problems, AFP reported.

Now, nearly a year and 16 surgeries later, Bajandar has his hands back.
 
"Bajandar's cure was a remarkable milestone in the history of medical science," Samanta Lal Sen, plastic surgery coordinator at Dhaka Medical College Hospital, told AFP. "We operated on him at least 16 times to remove the warts. The hands and feet are now almost fine."

Sen told AFP Bajandar could be the first person to be cured of the disease — though it's possible the warts could grow back. The condition killed an Indonesian man last year, AFP reported.

Bajandar, now 27, will endure a few more procedures, Sen told CNN, but they are more for "beautification."

Photos of Bajandar post-surgery show his limbs in thick bandages, but underneath his once-consumed hands now have thin, individual fingers free of the discolored growths. He can eat and write on his own, CNN reported.

"I never thought I would ever be able to hold my kid with my hands," Bajandar told AFP. "Now I feel so much better, I can hold my daughter in my lap and play with her. I can't wait to go back home."

[lilit]

da, da, to je uglavnom HPV 5, 8 ili 14 al u kombinaciji sa određenim genetskim mutacijama. sreća pa je retko.

btw, svakom bih preporučila da se vakciniše protiv HPV-a (i devojčice i dečaci). jeste da su unutra samo tipovi 6, 11, 16, 18, 31, 33, 45, 52, and 58 (hm, "samo" ), od kojih prva dva imaju nizak onkogeni potencijal i više su kondilomska i bradavična maltretaža, a ostali su high-oncogenic, al vakcina definitivno stvara zaštitu i protiv nekih tipova HPV-a koji nisu u vakcini.

[Meho Krljic]

US woman dies of infection resistant to all 26 available antibiotics

Miami (AFP) - A US woman has died from an infection that was resistant to all 26 available antibiotics, health officials said this week, raising new concerns about the rise of dangerous superbugs.
The woman, who was in her 70s, died in Nevada in September, and had recently been hospitalized in India with fractured leg bones, the US Centers for Disease Control and Prevention reported.
The cause of death was sepsis, following infection from a rare bacteria known as carbapenem-resistant Enterobacteriaceae (CRE), which is resistant to all antibiotics available in the United States.
The specific strain of CRE, known as Klebsiella pneumoniae, was isolated from one of her wounds in August.
Tests were negative for the mcr-1 gene -- a great concern to health experts because it makes bacteria resistant to the antibiotic of last resort, colistin.
It was unclear how the woman's infection acquired resistance.
Experts said she had been treated repeatedly in India during the last two years for a femur fracture and hip problems, most recently in June 2016.
Once the bacteria was identified in Nevada, the patient was isolated to prevent the infection from spreading in the hospital.
Postmortem tests showed her infection might have responded to a treatment called fosfomycin, which is not approved in the United States.
Paul Hoskisson, a researcher at the University of Strathclyde, in Scotland, said that several European countries, including Britain, license fosfomycin for intravenous use in such cases.
"This is important because we are seeing increasing numbers of drug-resistant infections, and this is one of the first cases for Klebsiella where no drug options were open to the medical staff."
Multi-drug-resistant Klebsiella pneumoniae has been described by the World Health Organization as "an urgent threat to human health."
According to Nick Thomson, leader of the bacterial genomics and evolution group at the Wellcome Trust Sanger Institute in England, this bacteria is likely to become more and more resistant.
"The report highlights international travel and treatment overseas as a feature in the introduction of this pan-resistant isolate into the USA," he said.
"Since we live in such an interconnected society, this is important because this isolate represents a truly untreatable infection" which leaves health-care professionals with few options but to seek to prevent further transmission.
Laura Piddock, a professor of microbiology at the University of Birmingham, said the case shows that doctors "need the flexibility to use antibiotics licensed for use in other countries and shown to be active in the laboratory against the patient's infecting bacterium."

A i ovo nije loše:

Your Appendix May Not Be Useless After All

The appendix, notorious for its tendency to become inflamed or even rupture, has historically been viewed as a vestigial organ with no real function. But new research supports the idea that the appendix may indeed serve a purpose: to protect beneficial bacteria living in the gut.
Heather F. Smith, PhD, associate professor at Midwestern University Arizona College of Osteopathic Medicine, has studied the evolution of gastrointestinal traits across different animal species. Her new research, published in the journal Comptes Rendus Palevol, analyzed the presence or absence of an appendix in 533 different mammals.
She found that the appendix evolved independently in different genetic "trees," more than 30 separate times. Furthermore, the appendix almost never disappeared from a lineage once it appeared. This suggests that the organ remains for a reason, she says—an adaptive purpose.
Smith and her co-authors—from Duke University Medical Center, the University of Stellenbosch in South Africa and the Muséum National d'Histoire Naturelle in France—were able to reject several previous hypotheses that the appendix may be linked to dietary or environmental factors. But they did make one interesting discovery: Species who had an appendix tend to have higher concentrations of lymphoid tissue in their cecum, a pouch that connects the small and large intestine.
This type of tissue can play a role in immunity, and can also stimulate growth of healthy gut bacteria. So it makes sense, Smith says, that the appendix actually serves as a "safe house" for these beneficial bugs.
This study isn't the first to suggest that the appendix may play this type of role. The "safe house" idea was first raised by a 2007 study, which inspired Smith to question whether the appendix had evolved to serve this function in humans and other mammals—a theory that now appears quite likely.
So what does this mean for people who have had their appendix removed? Luckily, not much. "In general, people who have had an appendectomy tend to be relatively healthy and not have any major detrimental effects," Smith says. (She herself had hers out at age 12.)
Some studies have shown, however, that people without an appendix may have slightly higher rates of infection than those with a functioning organ. "It may also take them slightly longer to recover from illness, especially those in which the beneficial gut bacteria has been flushed out of the body," Smith added.
In a broader sense, Smith says that research on the appendix has provided "another line of evidence against over-sanitizing and excessive hygiene." Because this organ is full of immune tissue, she says, one of the leading causes of appendicitis has to do with poorly developed immunity.
"Exposure to pathogens and infectious agents, like bacteria and viruses, is important for the normal development processes of the immune system," Smith says. Without this exposure, development can be suppressed and the immune system can become hypersensitive—a hypothesis often used to explain illnesses like asthma and allergies.
More research in this area may help doctors address the organ's most well known problem. "As treatments are developed for other autoimmune disorders and responses, it's certainly possible that something similar may be developed for treating appendicitis," she says.

Mada komentari na ovaj drugi članak... Počinjem da mislim da je davanje narodu platforme da se izrazi bila zaista loša, destruktivna ideja 

[lilit]

ovo prvo bi trebalo da ima naslov: only in usa

što se tiče slepog "no real function aaaaaaaaaaaaaaa" creva i "may not be useless after all"?????
(FOTO)

[Meho Krljic]

Prvo su došli po komarce, al mi nismo reagovali jer nismo komarci, onda sad dolaze po miševe i... Lilita, REAGUJ, ŽENO!!!

First Gene Drive in Mammals Could Aid Vast New Zealand Eradication Plan

Scientists working in coördination with a U.S. conservation group say they've established an evolution-warping technology called a "gene drive" in mammals for the first time and could use it to stamp out invasive rodents ravaging seabirds on islands.
Gene-drive technology, so far demonstrated only in insects and yeast, is a powerful way of biasing the inheritance of DNA such that wild animals can be genetically altered as they reproduce, including to cause a population crash.
Now two scientific teams—one in Australia and one in Texas—say they've genetically engineered the house mouse, Mus musculus, so that its genome also harbors genetic surprises that could be unleashed on wild populations. The modified rodents were born in the last two months and the results are still preliminary.
The effort to establish gene drives in mammals is being coördinated by Island Conservation, a hard-charging conservation group based in Santa Cruz, California, whose specialty is bombing small islands with rat poison in order to save endangered seabirds. Its motto is "preventing extinctions."
But poison doesn't work to extirpate rodents on larger islands or heavily populated ones. That's why the group thinks gene drives could be the "transformative technology" that allows it to extend its campaign to thousands more islands it says are infested. "We were looking for something really out of the box," says Karl Campbell, a program director at the nonprofit, which has plans to spent about $7 million a year to speed the technique toward an initial test on a remote island surrounded by miles of ocean, if authorities allow it.
Campbell says they are pursuing the creation of "daughterless" mice, which, due to a gene drive, are only able to have male offspring. The gender-biasing effect would drive down mouse populations on an island, possibly to zero if it proves effective.
The mice are an early glimpse of an idea being called "synthetic conservation," in which genetic engineering is viewed as a means to revive extinct animals, offer genetic refills for endangered species with shallow gene pools, or knock out invasive pests ravaging native plants and animals.
And rodents are high on the list of troublemakers. Brought by shipwrecks or sailors, they swarm over oceanic islands, imperiling native seabirds. While rats are the bigger problem, mice cause havoc, too. On Pacific islands, mice have been filmed gnawing on albatross chicks, which are defenseless against them.
The group's plans have divided ecologists, however, some of whom see a devil's bargain in the dizzying new power to modify nature. "Conservation means caring for the natural world, not reëngineering it," says Claire Hope Cummings, an environmental lawyer who says she dropped her support for Island Conservation over its gene-drive work.
Even proponents of gene-drive technology say it needs to be carefully studied and cautiously deployed, and also say it may not work as advertised. Last year, the U.S. National Academies advised a go-slow approach noting that "proof of concept in a few laboratories" isn't enough to "support a decision to release gene-drive organisms into the environment."
But it's hard not to see the potential. New Zealand, whose flightless birds were overrun starting in the 19th century by species brought by Westerners, this year announced plans to become "predator free" within 30 years by eliminating hundreds of millions of rats, possums, and weasels. The country's parliament has said that gene drives could be the very "breakthrough" that lets them achieve the goal.
"No holds barred? That's what we'd do. That would be phenomenal," says Campbell of clearing invasive predator species off New Zealand. "Then once we got through those, I don't see why you wouldn't be thinking about mainland areas" like slums or ecosystems which rats have also invaded, he says.
Daughterless mice
A man-made gene drive was first demonstrated in fruit flies only in 2015. Within a few months the concept had been extended to mosquitoes, and already the technology is viewed as promising enough to have landed $75 million from Bill Gates, whose foundation is betting that extinguishing mosquitoes could eradicate malaria from Africa.
So it was only a matter of time—less than two years, it turned out—before the technique was adapted to mammals.
The two groups of scientists coördinating their work with Island Conservation agreed to reveal the extent of their technical progress to MIT Technology Review, citing the need to develop powerful gene-drive technology in the open rather than behind closed doors.
"It's the perfect time to discuss the risks of the technology," says Paul Thomas, a mouse geneticist at the University of Adelaide, in South Australia. "We still have to see if it works at all. And it's not just showing it works, but how efficient and stable it is."
Thomas says he and his students created gene-drive mice using CRISPR, the powerful DNA-editing technology. To do it, Australians fashioned CRISPR into a "selfish gene" designed to transmit itself to nearly all of a mouse's offspring, rather than just half, as would be expected. To track its spread, they have also attached a fluorescent protein so mice who inherit it will glow red when you shine a black light on them.
With critics fretting over the possibility that a gene-drive organism could escape from the lab, Thomas says his lab has taken precautions to prevent a mishap, including designing safety features so the drive can't yet be transmitted to wild mice. When I spoke to him in January, Thomas said he was about to start breeding the first set of lab animals to determine if the drive works as predicted. This step, carried over a few generations, will take several months.
The other team is based at Texas A&M University and led by mouse geneticist David Threadgill, who says his lab has engineered first-generation "daughterless" mice. Some are now being bred to determine if the male-only trait is passed to future generations, as is hoped.
Instead of CRISPR, Threadgill's lab used a different strategy, harnessing a naturally occurring group of genes called the "t-complex." This genetic element also manages to spread itself selfishly by harming sperm that don't have it, and favoring those that do, so they end up fertilizing eggs and making more mouse pups. Versions of the t-complex are already present in many wild mice.
In order to make the mice daughterless, Threadgill's team introduced an additional modification. They attached to the t-complex an extra copy of Sry, a gene that is normally found on the Y chromosome and which determines whether a mammal turns out to be male. If the drive operates as intended—something that should be clear inside of a few weeks—more than nine in 10 mouse pups could inherit Sry and have male sex organs. Released in large enough numbers on an island, the daughterless rodents could, over the course of several months to a few years, result in a mouse population that is, so to speak, all Mickey and no Minnie. Then the mice would die out.
Rat Island
Island Conservation was formed in the 1990s, and its early exploits removing cats, goats, and even feral donkeys from Baja California are recounted in Rat Island, a book-length account of global island-clearing efforts, which by now have eradicated rodents from 500 islands.
The drawback is how conservation groups have relied on brodifacoum, a toxin said to be 100 times as potent as the rat poison warfarin. Rodents bleed to death after they eat it. So do any bald eagles and gulls unlucky enough to chomp the poisoned prey.
In theory, a gene drive is the perfect solution. It would affect only one species, and it is entirely painless. But some scientists caution that the technology may never work as planned. Coddled lab mice put on an island would be the first to get grabbed by a raptor. And females might be able to sniff out the gene drive, shunning certain males, or even develop resistance to it.  "I think there are actually a hell of a lot of things that could go wrong," says Neill Gemmell, a researcher at the University of Otago in New Zealand. "If you think you are just going to release things and they are going to eradicate for you, it's a big mistake."
Island Conservation, which initially said it planned to try an offshore test by 2020, has since backed away from that prediction, citing open-ended technical and regulatory questions.
That's not to say Gemmell isn't interested. In 2016, New Zealand's government formally launched "Predator Free 2050"—its ambitious plan to kill every rat, possum, and weasel across its 103,483-square-mile territory. Program documents call gene drives a "realistic prospect," and Gemmell is a part of a committee looking at the options.
Using a genetic assault along with poison and traps is probably the only way to ensure the eradication comes off "cheaply and quickly," says Gemmell, but the hurdles look daunting. Even if the drives work in mice, no one has ever before genetically engineered an opossum or a weasel. And what would a breeding center able to turn out thousands of GM possums a week even look like? What's more, because possums breed only once a year, it could take many years, or decades, for a gene drive to have its lethal effect. 
The use of gene drives won't be able to move forward without wide public support. And that could be difficult to win given how it is already dividing conservationists. Some groups, like Friends of the Earth, are deeply suspicious of any genetic engineering and call genes drives a "false solution to the real problem of biodiversity loss."
Cummings, the environmental lawyer who is also the author of a book critical of GMOs, says she's also alarmed by the plans to target female mice. "Daughterless anything is a problem," she says. "The whole 'eliminate the female' concept needs to be looked at philosophically and ethically." Cummings, who has listened to Island Conservation's arguments, says she's come to the conclusion that saving seabirds from rats is being used to "whitewash this technology, give it moral cover, when it might be the world's most dangerous bioweapon."
The two sides debated several times last year, most recently in December, at a meeting in Cancun of the UN Convention on Biodiversity, where activists including Friends of the Earth and ETC Group rallied about 170 civil society groups calling for a moratorium on gene drives. A letter signed by luminaries including the primatologist Jane Goodall warned that "genocidal genes" could have "consequences beyond our comprehension."
The effort to compel a ban fell short.

[lilit]

izgubio mi se altruizam u međuvremenu i za komarce i za glodare, a i kuga je teška bolest.
reagovah.

[Meho Krljic]

The dirty dozen: UN issues list of 12 most worrying bacteria

The World Health Organization has issued a list of the top dozen bacteria most dangerous to humans, warning that doctors are fast running out of treatment options.

In a press briefing on Monday, the U.N. health agency said its list is meant to promote the development of medicines for the most worrying drug-resistant bacteria, including salmonella and Staphylococcus aureus.

WHO's Marie-Paule Kieny said that if such priorities were left to market forces alone, "the new antibiotics we most urgently need are not going to be developed in time." She estimated that it would take up to a decade for new medications.

WHO said the most-needed drugs are for germs that threaten hospitals, nursing homes and among patients who need ventilators or catheters. The agency said the dozen listed resistant bacteria are increasingly untreatable and can cause fatal infections; most typically strike people with weakened immune systems.

At the top of WHO's list is Acinetobacter baumannii, a group of bacteria that cause a range of diseases from pneumonia to blood or wound infections.

WHO's list was developed in collaboration with the University of Tubingen in Germany and according to criteria assessed by international experts.

Among the experts' and WHO's most pressing concerns were how deadly the infections were, whether treatment required a long hospital stay and how many current medicines exist.

In recent years, health officials have detected a few patients resistant to colistin, the antibiotic of last resort. So far, doctors have been able to treat them with other drugs.

But experts worry that the colistin-resistant bacteria will spread their properties to other bacteria already resistant to more commonly used antibiotics, creating germs that can't be killed by any known drugs.

According to the U.S. Centers for Disease Control and Prevention, at least 23,000 people die each year in the U.S. from infections caused by resistant bacteria.

[lilit]

joj videla sam ovo pre neki dan i rado bih se bacila na staphylococcus aureus, neverovatno je koliko je to slatka a potencijalno opasna bakterijica.
nigde hlamidijena listi.

[Meho Krljic]

Drastično opada broj dece zaštićene MMR vakcinom

[lilit]

meni nekako mnogo teško da poverujem da se u 2017. godini natežemo oko vakcina. uz "safe water", nijedna mera koju je primenila civilizacija nije uticala na smanjenje smrtnosti i rast populacije kao što je to uradilo uvođenje vakcinacije. uskoro će i ceo vek od uvođenja vakcina protiv tetanusa, difterije i velikog kašlja i ko danas ne vakciniše decu protiv te tri bolesti, u mom utopijskom društvu bi završio bar u zatvoru.
što se tiče mmr vakcine, ko god nije preležao posebno male boginje treba sigurno da se revakciniše (mehane, odmah! ). ko u odraslim godinama dobije male boginje nek računa da će imati problem s imunitetom bar 3 godine od prestanka bolesti. measles su jedini poznati virus koji je u stanju da uništi kompletnu prethodnu, godinama sticanu, imunološku memoriju.

[Meho Krljic]

Could diabetes spread like mad cow disease?

[lilit]

razmišljam sa kliničkog aspekta i nekako ne vidim što bi bilo ko pristao da se izloži injekciji ekstrakta pankreasa osobe obolele od dijabetesa (na stranu što nije ni etički da to radimo živim ljudima  ), a sumnjam i da ćemo taj pankreas nekad smatrati za delikates.
čak i da je prelazno, teško ćeš transfuzijom dobiti dovoljne količine za transmisiju.
u svakom slučaju, zanimljiv koncept i odlično dizajnirana studija.

btw, prioni su mi već evo 20 godina full fascinacija. obožavam ih skoro kao HIV.

[Meho Krljic]

Znam ja šta voliš 

[lilit]

... pomanjkanje empatije koja je za mene samo akademski koncept.

QQ, ovo ko da sam te ja rodila.
Moje najstarije dete pre neki dan izašlo s istom teorijom ("na nivou koncepta razumem empatiju, al je ne osećam"  ) i srce mi slomilo pošto mene empatija i za mašine povremeno razbije. 

[Meho Krljic]

Meet Paranthropus Boisei, The Ancient Hominin That Gave Humans Genital Herpes

[lilit]

moram da priznam da mi "prion-lajk-prenošenje-virusa" teorija nikako ne pije vodu. HSV1 i HSV2 ne mogu da prežive ni na dasci od WC šolje duže od nekoliko minuta, a da probiju HCl u želucu (jedem inficiranu hranu pa mi herpes dođe do genitalija) je već na ivici SF-a.
to da humanoidima paranthropus boisei nije bio privlačan mogu da prihvatim, al pretpostavljam da je bilo humanoida i humanoida, kao i danas h.sapiensa i h.sapiensa, e.g. ima nas raznih.
al ok, ko zna kakvi su ti herpesi nekad bili.

[Meho Krljic]

Da, možda je OG herpes bio surov i nezaustavljiv a ovaj današnji je njegov pitomiji potomak koji je mutirao u nešto manje zajebano pa je time i rašireniji...

[Meho Krljic]

E, da vidmo...


''Galenika'' prodata kompaniji ''Elius'' iz Luksemburga

[lilit]

hm. bilo bi zanimljivo videti šta je prodato, al raspitaću se pa se javljam.

[Meho Krljic]

Prvo su došli po komarce

I saće im jebu kevu:

US government approves 'killer' mosquitoes to fight disease



Ameriko, nadamo se da si sigurna da znaš šta radiš.

[lilit]

ne znaju.
odnosno, ne znaju dovoljno o mehanizmu kako wolbachia utiče na reprodukciju virusa koji koriste komarce kao brodove za naseljavanje novih svetova hostova.
naravno, razne wolbachije (vrlo simpatična bakterija, po razvojnom ciklusu slična hlamidiji), razni virusi, razni komarci.
ideja im je lepa i naučno potkovana, o tome se razmišlja više od 40 godina. za zika virus će da odradi posao, direktno će da utiče na smanjenje reprodukcije inficiranih komaraca.
problem je što je pokazano da wolbachia povećava reprodukciju west nile virusa u komarcima i trebalo bi što pre razumeti zašto i kako.

[Meho Krljic]

HPV is causing an oral cancer epidemic in men by outwitting natural defenses

[lilit]

da, da, oralni kancer je rastući problem, cifre su zabrinjavajuće. obično ga detektuju tek kad odmakne i zato se od pre nekoliko godina preporučuje i vakcinacija dečaka, pored ranije uvedene vakcinacije devojčica. trenutno se koristi vakcina koja se zove Gardasil 9 (pokriva 9 tipova HPV od kojih su 7 high-risk onkogeni) i daju se dve doze u razmaku od 6 meseci. u austriji je besplatna za decu od 9-12 godina, do 15. se može dobiti po sniženoj ceni, posle toga košta oko 180 evra/dozi.

[Petronije]

Lilit, sećaš li se u kojoj temi si pisala o vakcinaciji i malim boginjama?

Sent from my Le X626 using Tapatalk

[lilit]

na raznim, kad god sam stigla. 

šta konkretno?

[Petronije]

Pa hteo bih da iščitam sve, ali najviše me zanimalo oko vakcinacije, znam da si spominjala koje su generacije vakcinisane a koje ne.

Sent from my Le X626 using Tapatalk

[Meho Krljic]

Evo, da pomognem, ima to ovde:

http://www.znaksagite.com/diskusije/index.php?topic=5367.msg696580;topicseen#msg696580


A sad druga tema:

Amazon has a fix for Alexa's creepy laughs

[Petronije]

Hvala, našao sam šta me je zanimalo.

Sent from my Le X626 using Tapatalk

[lilit]

ne mogu ni ja sve da nađem, verovatno ne searchujem kako treba.

evo ukratko:

Monovalentna vakcina protiv virusa malih boginja (ono prvo M u MMR kombinovanoj trovalentnoj vakcini) je puštena na svetsko tržište 1963. godine (FOTO 1! )



Jugoslavija je uvezla vakcinu 1971, tu se nešto čekalo oko registracije, posle smo imali slučaj velikih boginja (1972. Variola vera) i u to doba fokus je bio na vakcinaciji protiv velikih boginja. Moj, neobjektivni , utisak je da je vakcinacija protiv malih boginja krenula ozbiljnije tek 1973. godine. Preporuka je bila – jedna doza, što se u kasnijim decenijama pokazalo kao nedovoljno (i to nije ništa neobično – kliničke studije se rade decenijama da bi se neki nalaz potvrdio/opovrgao).

Pokazano je da jedna doza ne daje dugotrajni imunitet, sad je preporuka 2 doze + testiranje antitela (da se vidi kako stoji imunitet) na svakih 10 godina.
Ja sam to radila pre neki dan (FOTO 2! )



MMR vakcina (trovalentna: male boginje, zauške i crvenka) je u Srbiju ušla 1993. godine a sa revakcinacijom se krenulo 1996. Koliko MMR-a je Torlak uvozio u doba sankcija i koji imbecili su o tome odlučivali, ne bih ni da se prisećam. Ali sigurna sam da Srbija in general nije revakcinisana, a svi rođeni pre 1973. godine (a nisu preležali male boginje) nemaju ni bazični nivo zaštite.

U Austriji je od ove godine, a opet na osnovu kliničkih studija, preporuka da se prva doza MMR vakcine daje kad beba navrši 9 meseci, a druga doza 3 meseca nakon toga.  Dalje, u slučaju da se MMR vakcina (iz bilo kojih razloga) aplicira nakon navršene prve godine života, druga doza u tom slučaju ide 4 nedelje nakon prve imunizacije. U Srbiji se i dalje druga doza prima u školi.

Dodatne preporuke: ko god nema dokumentovano (i) primljene 2 doze i dokaz u vakcinalnom kartonu da je vakcinisia ili (ii) titar antitela) da je imun, preporuka je da se vakciniše.           

[lilit]

fala meho! hm, u tom postu je koncentrisana sva moja netolerancija! lepo za videti!

petronije, nadam se da ti je ovo OK. sad tek videh da si našao šta si tražio.

[lilit]

A sad druga tema:

Amazon has a fix for Alexa's creepy laughs

sad sam je pitala.
kaže: - sure I can laugh. hihi.
moja alexa je kjuti!

pitala sam dodatno: - alexa, do you know meho krljić?
kaže da joj nisi poznat.



dobro jutro sagitice.

[Meho Krljic]

S obzirom da je to moje ime prvi put upotrebljeno za Amazon rivju, zaista je uvredljivo da me Alexa ne poznaje. But oh well... Izdržao sam i gore napade u životu!!!!!!!!!!!!!!

[Mica Milovanovic]

Našla si njega... Mene mora da poznaje... 

[Petronije]

Odlično lilit, hvala i tebi, ovo je i više nego što treba. 

[lilit]

Našla si njega... Mene mora da poznaje... 

kaže nein. imam i video snimak!

petronije, 

[Mica Milovanovic]

Pa kad pričaš sa njom na njemačkom. U, Radosave...


https://www.youtube.com/watch?v=sPX8DlgMtyg

[lilit]

sad malo back to research.

How 30 years of research has halted HPV and cut cancer
Including a top 5 of the latest studies published in Papillomavirus Research that are contributing to ending HPV transmission

https://www.elsevier.com/life-sciences/journals/how-30-years-of-research-has-halted-hpv-and-cut-cancer

ogroman problem je što se često common knowledge teško prihvata i od profesionalaca, pa je kontinuirana obuka lekara neophodna. npr. u srbiji već ima pedijatara koji trudnim ženama govore da ne vakcinišu drugo dete (ide u obdanište)  MMR vakcinom jer su trudne. jes da je vakcina živa, al jes i da se decenijama zna da je MMR vakcina protiv malih boginja tako umrtvljena da ne "shedduje" virus na okolinu. a to bi trebalo da je elementary knowledge i za lekare opšte prakse, i za pedijatre, i za ginekologe, etc.

We know that in spite our 3,000 papers per year, many professionals have not read them. We have to update medical professionals to explain HPV prevention to hundreds of thousands of people in many different cultures: to the family that is worrying about vaccinating their children, to people who want HPV screening over and above the Pap smear, and to help people understanding what we can do if they are found as HPV positive. We know how to prevent several important cancers in men and women. People should be aware that these viruses exist, and that there are preventative opportunities.

[tomat]

https://pescanik.net/dobrodosli-u-austriju/

[Aco Popara Zver]

Odlično, samo jedna stvar: to nije spoj neoliberalizma i nacionalizma no čisti neoliberalizam.

Dolaze musoliniji i raduli, i svi su swatki prije dolaska na funkciju.

[mac]

Da li ćeš spominjati Radulovića i kad neko drugi dođe na čelo DJB?

[Aco Popara Zver]

Misliš kad Lude dođe?

Naravno! Radul siva eminencia!

[lilit]

https://pescanik.net/dobrodosli-u-austriju/

kako stvari sad izgledaju, nije čak ni previše loše što su na vlast došli ti koji su došli jer posle ovog mandata imaćemo opet socijaliste narednih 50 godina. to kažu i glasači ÖVP-a na prošlim izborima.

[lilit]

ovo ću ovde da zakačim, za slučaj da kome zatreba.

uputstvo kako objasniti komšiji, kolegi, detetu... zašto treba da se vakciniše:
https://thenib.com/vaccines-work-here-are-the-facts-5de3d0f9ffd0?t=default

jeste da je 2018. i da je za neverovati da danas moramo da se ubeđujemo oko benefita koje su nam vakcine pružile, al izgleda da ćemo, kao čovečanstvo, morati da startujemo od osnova.

[Ana]

Hvala! Podelila.
Ali, za mnoge naše antivaksere treba i prevesti na srpski. I ispisati ćirilicom.

[Dybuk]

Bojim se da ne vredi, jer ljudi imaju uverenja, i od njih ne odustaju.
Nisu to uvek prosti, neobavešteni ili neobrazovani ljudi, ne. Jednostavno, to je sklonost da veruješ u teorije zavere i kvit.

[Truba]

Zašto nema topika bulshit reagiraj...tojest black swan
Ako ga sad neko pokrene nije to to...isforsirano